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PhD, University of Louisville




THE ROLE OF MITOFUSINS IN ENDOTHELIAL MITOCHONDRIAL SIGNALING AND ENDOTHELIAL CELL FUNCTION
06/01/2016 - 05/31/2018 (PI)
NIH/National Diabetes & Digestive & Kidney Diseases
5K01DK097414-05

THE ROLE OF MITOFUSINS IN ENDOTHELIAL MITOCHONDRIAL SIGNALING AND ENDOTHELIAL CELL FUNCTION
08/01/2013 - 05/31/2018 (PI)
NIH/National Heart, Lung, and Blood Institute
1K01DK097414-01A1

THE ROLE OF MITOFUSINS IN ENDOTHELIAL MITOCHONDRIAL SIGNALING AND ENDOTHELIAL CELL FUNCTION
06/01/2014 - 05/31/2016 (PI)
NIH/National Diabetes & Digestive & Kidney Diseases
5K01DK097414-03



Publications listed below are automatically derived from MEDLINE/PubMed and other sources, which might result in incorrect or missing publications. Faculty can login to make corrections and additions.

  1. Papanicolaou KN, Kikuchi R, Ngoh GA, Coughlan KA, Dominguez I, Stanley WC, Walsh K. Mitofusins 1 and 2 are essential for postnatal metabolic remodeling in heart. Circ Res. 2012 Sep 28; 111(8):1012-26.View Related Profiles. PMID: 22904094; PMCID: PMC3518037.
     
  2. Ngoh GA, Papanicolaou KN, Walsh K. Loss of mitofusin 2 promotes endoplasmic reticulum stress. J Biol Chem. 2012 Jun 8; 287(24):20321-32. PMID: 22511781; PMCID: PMC3370214; DOI: 10.1074/jbc.M112.359174;.
     
  3. Facundo HT, Brainard RE, Watson LJ, Ngoh GA, Hamid T, Prabhu SD, Jones SP. O-GlcNAc signaling is essential for NFAT-mediated transcriptional reprogramming during cardiomyocyte hypertrophy. Am J Physiol Heart Circ Physiol. 2012 May 15; 302(10):H2122-30. PMID: 22408028; PMCID: PMC3362113; DOI: 10.1152/ajpheart.00775.2011;.
     
  4. Papanicolaou KN, Ngoh GA, Dabkowski ER, O'Connell KA, Ribeiro RF, Stanley WC, Walsh K. Cardiomyocyte deletion of mitofusin-1 leads to mitochondrial fragmentation and improves tolerance to ROS-induced mitochondrial dysfunction and cell death. Am J Physiol Heart Circ Physiol. 2012 Jan 1; 302(1):H167-79.View Related Profiles. PMID: 22037195; PMCID: PMC3334239; DOI: 10.1152/ajpheart.00833.2011;.
     
  5. Lugus JJ, Ngoh GA, Bachschmid MM, Walsh K. Mitofusins are required for angiogenic function and modulate different signaling pathways in cultured endothelial cells. J Mol Cell Cardiol. 2011 Dec; 51(6):885-93.View Related Profiles. PMID: 21839087; PMCID: PMC3208756; DOI: 10.1016/j.yjmcc.2011.07.023;.
     
  6. Papanicolaou KN, Khairallah RJ, Ngoh GA, Chikando A, Luptak I, O'Shea KM, Riley DD, Lugus JJ, Colucci WS, Lederer WJ, Stanley WC, Walsh K. Mitofusin-2 maintains mitochondrial structure and contributes to stress-induced permeability transition in cardiac myocytes. Mol Cell Biol. 2011 Mar; 31(6):1309-28.View Related Profiles. PMID: 21245373; PMCID: PMC3067905; DOI: 10.1128/MCB.00911-10;.
     
  7. Watson LJ, Facundo HT, Ngoh GA, Ameen M, Brainard RE, Lemma KM, Long BW, Prabhu SD, Xuan YT, Jones SP. O-linked ß-N-acetylglucosamine transferase is indispensable in the failing heart. Proc Natl Acad Sci U S A. 2010 Oct 12; 107(41):17797-802. PMID: 20876116; PMCID: PMC2955091; DOI: 10.1073/pnas.1001907107;.
     
  8. Ngoh GA, Watson LJ, Facundo HT, Jones SP. Augmented O-GlcNAc signaling attenuates oxidative stress and calcium overload in cardiomyocytes. Amino Acids. 2011 Mar; 40(3):895-911. PMID: 20798965; PMCID: PMC3118675; DOI: 10.1007/s00726-010-0728-7;.
     
  9. Ngoh GA, Facundo HT, Zafir A, Jones SP. O-GlcNAc signaling in the cardiovascular system. Circ Res. 2010 Jul 23; 107(2):171-85. PMID: 20651294; PMCID: PMC2919351; DOI: 10.1161/CIRCRESAHA.110.224675;.
     
  10. Schaller S, Paradis S, Ngoh GA, Assaly R, Buisson B, Drouot C, Ostuni MA, Lacapere JJ, Bassissi F, Bordet T, Berdeaux A, Jones SP, Morin D, Pruss RM. TRO40303, a new cardioprotective compound, inhibits mitochondrial permeability transition. J Pharmacol Exp Ther. 2010 Jun; 333(3):696-706. PMID: 20215409; DOI: 10.1124/jpet.110.167486;.
     
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This graph shows the total number of publications by year, by first, middle/unknown, or last author.



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