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CED-4 CARD domain residues can modulate non-apoptotic neuronal regeneration functions independently from apoptosis.Academic Article Why?
Novel DLK-independent neuronal regeneration in Caenorhabditis elegans shares links with activity-dependent ectopic outgrowth.Academic Article Why?
Gabel, ChristopherPerson Why?
Modulation of Cellular Metabolism to Maximize Neuronal RegenerationGrant Why?
Neuronal regeneration in C. elegans requires subcellular calcium release by ryanodine receptor channels and can be enhanced by optogenetic stimulation.Academic Article Why?
The core apoptotic executioner proteins CED-3 and CED-4 promote initiation of neuronal regeneration in Caenorhabditis elegans.Academic Article Why?
Neural plasticity after peripheral nerve injury and regeneration.Academic Article Why?
Test-retest reliability of fMRI during nonverbal semantic decisions in moderate-severe nonfluent aphasia patients.Academic Article Why?
Chung, SamuelPerson Why?
Doublecortin-Like Kinases Promote Neuronal Survival and Induce Growth Cone Reformation via Distinct Mechanisms.Academic Article Why?
Sun, LinPerson Why?
Apicco, DanielPerson Why?
Mcloed, MelissaPerson Why?
Role of hypoxia inducible factor-1 alpha pathway in bone regeneration.Academic Article Why?
The histopathology of neuronal degeneration and plasticity in Alzheimer's diseaseAcademic Article Why?
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