Search Results (17)
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MatchTypeWhy
Basic fibroblast growth factor enhances axonal sprouting after cortical injury in rats.Academic Article Why?
Cell based therapy reduces secondary damage and increases extent of microglial activation following cortical injury.Academic Article Why?
Mesenchymal derived exosomes enhance recovery of motor function in a monkey model of cortical injury.Academic Article Why?
Recovery of Motor Function Impaired by Cortical Injury: An Exploratory Comparison of the influences of Four Different Post-injury Treatments on Processes Affecting NeuroplasticityGrant Why?
Recovery of Motor Function Impaired by Cortical Injury: An Exploratory Comparison of the influences of Four Different Post-injury Treatments on Processes Affecting NeuroplasticityGrant Why?
A method for assessing recovery of fine motor function of the hand in a rhesus monkey model of cortical injury: an adaptation of the Fugl-Meyer Scale and Eshkol-Wachman Movement Notation.Academic Article Why?
Inosine enhances recovery of grasp following cortical injury to the primary motor cortex of the rhesus monkey.Academic Article Why?
Extracellular vesicles from mesenchymal stem cells reduce microglial-mediated neuroinflammation after cortical injury in aged Rhesus monkeys.Academic Article Why?
The efficacy of curcumin to facilitate recovery of function in a rhesus monkey model of cortical injuryGrant Why?
The efficacy of curcumin to facilitate recovery of function in a rhesus monkey model of cortical injuryGrant Why?
Concussive injury before or after controlled cortical impact exacerbates histopathology and functional outcome in a mixed traumatic brain injury model in mice.Academic Article Why?
Mild traumatic brain injury is associated with reduced cortical thickness in those at risk for Alzheimer''s disease.Academic Article Why?
Cortical thinning in former professional soccer players.Academic Article Why?
Sex differences in the effect of progesterone after controlled cortical impact in adolescent mice: a preliminary study.Academic Article Why?
Supply-demand mismatch transients in susceptible peri-infarct hot zones explain the origins of spreading injury depolarizations.Academic Article Why?
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