Click the Why column to see why an item matched the search.
| Match | Type | Why |
|---|
| Airway basal stem cells reutilize the embryonic proliferation regulator, Tgfß-Id2 axis, for tissue regeneration. | Academic Article |
Why?
|
| TGFß-induced fibroblast activation requires persistent and targeted HDAC-mediated gene repression. | Academic Article |
Why?
|
| APOE4 impairs the microglial response in Alzheimer''s disease by inducing TGFß-mediated checkpoints. | Academic Article |
Why?
|
| Distinct polarity cues direct Taz/Yap and TGFß receptor localization to differentially control TGFß-induced Smad signaling. | Academic Article |
Why?
|
| Poly(I:C) drives type I IFN- and TGFß-mediated inflammation and dermal fibrosis simulating altered gene expression in systemic sclerosis. | Academic Article |
Why?
|
| Externalized histones fuel pulmonary fibrosis via a platelet-macrophage circuit of TGFß1 and IL-27. | Academic Article |
Why?
|
| TGFß signaling regulates lipogenesis in human sebaceous glands cells. | Academic Article |
Why?
|
| TGFß signaling underlies hematopoietic dysfunction and bone marrow failure in Shwachman-Diamond Syndrome. | Academic Article |
Why?
|
| TGFß/Smad3 regulates proliferation and apoptosis through IRS-1 inhibition in colon cancer cells. | Academic Article |
Why?
|
| Vitamin D3 Treatment Influences PGE2 and TGFß in Normal and Increased Breast Cancer Risk Women. | Academic Article |
Why?
|
| Abrogated transforming growth factor beta receptor II (TGFßRII) signalling in dendritic cells promotes immune reactivity of T cells resulting in enhanced atherosclerosis. | Academic Article |
Why?
|
| Activated Endothelial TGFß1 Signaling Promotes Venous Thrombus Nonresolution in Mice Via Endothelin-1: Potential Role for Chronic Thromboembolic Pulmonary Hypertension. | Academic Article |
Why?
|
| Divergent signaling pathways cooperatively regulate TGFß induction of cysteine-rich protein 2 in vascular smooth muscle cells. | Academic Article |
Why?
|
| Estrogen receptor a or ß loss in the colon of Min/+ mice promotes crypt expansion and impairs TGFß and HNF3ß signaling. | Academic Article |
Why?
|
| Few Smad proteins and many Smad-interacting proteins yield multiple functions and action modes in TGFß/BMP signaling in vivo. | Academic Article |
Why?
|