Nurselin Ates, PhD
Postdoctoral Associate
Boston University Chobanian & Avedisian School of Medicine



Publications listed below are automatically derived from MEDLINE/PubMed and other sources, which might result in incorrect or missing publications. Faculty can login to make corrections and additions.

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  1. Inci OK, Leal AF, Ates N, Súarez DA, Espejo-Mojica AJ, Alméciga-Diaz CJ, Seyrantepe V. Yeast-Produced Human Recombinant Lysosomal ß-Hexosaminidase Efficiently Rescues GM2 Ganglioside Accumulation in Tay-Sachs Disease. J Pers Med. 2025 May 10; 15(5). PMID: 40423067; PMCID: PMC12113087; DOI: 10.3390/jpm15050196;
     
  2. Basirli H, Ates N, Seyrantepe V. Imbalance in redox homeostasis is associated with neurodegeneration in the murine model of Tay-Sachs disease. Mol Biol Rep. 2025 Mar 05; 52(1):282. PMID: 40042748; DOI: 10.1007/s11033-025-10380-y;
     
  3. Sengul T, Can M, Ates N, Seyrantepe V. Autophagic flux is impaired in the brain tissue of Tay-Sachs disease mouse model. PLoS One. 2023; 18(3):e0280650. PMID: 36928510; PMCID: PMC10019743; DOI: 10.1371/journal.pone.0280650;
     
  4. Demir SA, Timur ZK, Ates N, Martínez LA, Seyrantepe V. GM2 ganglioside accumulation causes neuroinflammation and behavioral alterations in a mouse model of early onset Tay-Sachs disease. J Neuroinflammation. 2020 Sep 20; 17(1):277. PMID: 32951593; PMCID: PMC7504627; DOI: 10.1186/s12974-020-01947-6;
     
  5. Seyrantepe V, Demir SA, Timur ZK, Von Gerichten J, Marsching C, Erdemli E, Oztas E, Takahashi K, Yamaguchi K, Ates N, Dönmez Demir B, Dalkara T, Erich K, Hopf C, Sandhoff R, Miyagi T. Murine Sialidase Neu3 facilitates GM2 degradation and bypass in mouse model of Tay-Sachs disease. Exp Neurol. 2018 Jan; 299(Pt A):26-41. PMID: 28974375; DOI: 10.1016/j.expneurol.2017.09.012;
     

This graph shows the total number of publications by year, by first, middle/unknown, or last author.

Bar chart showing 5 publications over 4 distinct years, with a maximum of 2 publications in 2025

YearPublications
20171
20201
20231
20252

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