Search Results (33)
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MatchTypeWhy
Cellular activation triggered by the autosomal dominant polycystic kidney disease gene product PKD2.Academic Article Why?
Loss of PKD1 and loss of Bcl-2 elicit polycystic kidney disease through distinct mechanisms.Academic Article Why?
MRI isolation of infected renal cyst in autosomal dominant polycystic kidney disease.Academic Article Why?
Renal and extrarenal autosomal dominant polycystic kidney disease.Academic Article Why?
Polycystic Kidney Disease and Other Inherited Tubular DisordersAcademic Article Why?
Polycystic Kidney DiseasesConcept Why?
Polycystic Kidney, Autosomal DominantConcept Why?
Polycystic Kidney, Autosomal RecessiveConcept Why?
Research Fellowship--Polycystic Kidney DiseaseGrant Why?
An integrated genetic and physical map of the 650-kb region containing the congenital polycystic kidney (cpk) locus on mouse chromosome 12.Academic Article Why?
Inhibition of HER-2(neu/ErbB2) restores normal function and structure to polycystic kidney disease (PKD) epithelia.Academic Article Why?
Cohen, HerbertPerson Why?
The polycystic kidney disease 1 (Pkd1) gene is required for the responses of osteochondroprogenitor cells to midpalatal suture expansion in mice.Academic Article Why?
A human PKD1 transgene generates functional polycystin-1 in mice and is associated with a cystic phenotype.Academic Article Why?
Comparison of Pkd1-targeted mutants reveals that loss of polycystin-1 causes cystogenesis and bone defects.Academic Article Why?
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